Because it is released by a motor neuron and not an endocrine gland. Hormones are released only by glands.
The release of acetylcholine at the neuromuscular junction is not considered a hormonal event because it involves the transmission of a neurotransmitter (acetylcholine) between nerve cells and muscle cells, which occurs locally and rapidly. Hormones are typically produced by endocrine glands, circulated through the bloodstream, and have slower and longer-lasting effects on target cells throughout the body.
Acetylcholine is the chemical that transmits signals across the neuromuscular junction. It binds to receptors on the muscle cell membrane, leading to muscle contraction.
Curare acts by blocking nicotinic acetylcholine receptors at the neuromuscular junction, preventing the binding of acetylcholine to these receptors. This leads to muscle paralysis since acetylcholine is unable to initiate muscle contraction.
Ach (acetylcholine) is a neurotransmitter that is released by motor neurons at the neuromuscular junction. It binds to receptors on muscle cells, leading to muscle contraction.
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ACETYLCHOLINE
Acetylcholine is the chemical that transmits signals across the neuromuscular junction. It binds to receptors on the muscle cell membrane, leading to muscle contraction.
Curare acts by blocking nicotinic acetylcholine receptors at the neuromuscular junction, preventing the binding of acetylcholine to these receptors. This leads to muscle paralysis since acetylcholine is unable to initiate muscle contraction.
Ach (acetylcholine) is a neurotransmitter that is released by motor neurons at the neuromuscular junction. It binds to receptors on muscle cells, leading to muscle contraction.
it has caca
No, acetylcholine is not only found at the neuromuscular junction. It is a neurotransmitter that is also found in the brain and is involved in various functions such as memory, learning, and regulating the autonomic nervous system.
Acetylcholine is the primary neurotransmitter released at the neuromuscular junction. It is responsible for transmitting signals from motor neurons to muscle fibers, leading to muscle contraction.
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Acetylcholine is the primary chemical transmitter released at the neuromuscular junction. It binds to acetylcholine receptors on the muscle cell membrane, leading to muscle contraction.
Depolarizing neuromuscular blocking drugs, like succinylcholine, cause initial depolarization of the muscle cell membrane followed by prolonged depolarization. Non-depolarizing drugs, like rocuronium, block the action of acetylcholine at the neuromuscular junction without causing depolarization.
Acetylcholine is released by axon terminals of the motor neuron.
Β binding of acetylcholine to membrane receptors on the sarcolemma