GROWTH hormone
growth hormone
Growth hormone (GH) generally promotes growth and development, particularly affecting the proliferative activity of the growth plate, or epiphyseal plate, in long bones. Increased levels of growth hormone can stimulate the proliferation of chondrocytes, which are responsible for cartilage growth in this area. Therefore, growth hormone is unlikely to cause decreased proliferation at the epiphyseal plate; rather, it typically enhances it, leading to increased linear bone growth. However, excessive GH can lead to conditions like acromegaly, which may affect bone growth patterns.
This type of stimulus is called hormonal stimulation. It occurs when one hormone signals another endocrine gland to release its own hormone to regulate physiological processes in the body.
Adrenal gland activity is evaluated by the plasma level of cortisol hormone.
There are many hormones which stimulate activity in the ovaries.The two main ones are FSH (follicle-stimulating hormone) and LH (luteinizing hormone). FSH triggers the growth of an ovum, and LH is involved in causing ovulation (the release of the ovum/egg).
growth hormone
During early childhood, growth hormone is the most important factor stimulating growth at the epiphyseal plate. This hormone, produced by the pituitary gland, promotes the proliferation of chondrocytes in the growth plate, leading to increased bone length. Additionally, factors such as nutrition and physical activity can support the effects of growth hormone by ensuring that the body has the necessary resources for optimal growth.
human growth hormone
growth hormone
In bone formation, a deficiency of growth hormone will cause DECREASED PROLIFERATION OF THE EPIPHYSEAL PLATE CARTILAGE.
estrogen directly causes the epiphyseal plates to close, testosterone is converted to estradiol (estrogen) by aromatase, therefore testosterone causes the epiphyseal plates to close indirectly
erythropoietin
calcium
Actions of Growth Hormone.
Growth hormone (GH) generally promotes growth and development, particularly affecting the proliferative activity of the growth plate, or epiphyseal plate, in long bones. Increased levels of growth hormone can stimulate the proliferation of chondrocytes, which are responsible for cartilage growth in this area. Therefore, growth hormone is unlikely to cause decreased proliferation at the epiphyseal plate; rather, it typically enhances it, leading to increased linear bone growth. However, excessive GH can lead to conditions like acromegaly, which may affect bone growth patterns.
The hormone responsible for terminating growth at the epiphyseal plate in both men and women as they end adolescence is estrogen. Estrogen plays a key role in promoting the closure of the growth plates, leading to the cessation of longitudinal bone growth.
The Parathyroid hormone has a decrease in plasma calcium. It uses a Pentagastrin as stimulation for its release.