Wiki User
β 8y agoWhen acetylcholine binds to receptors at the motor plate, this binding opens ligand-gated ion channels on the motor end plate, ions diffuse through the open ligand gated ion channels, and the flow of ions causes the motor end plate to reach threshold and an action potential is generated at the motor end plate.
Wiki User
β 8y agoWhen acetylcholine binds to receptors at the motor plate, it triggers the opening of ion channels in the muscle membrane. This allows sodium ions to flow into the muscle cell, leading to depolarization of the membrane and initiation of a muscle action potential. Subsequently, this action potential travels along the muscle cell membrane, leading to muscle contraction.
Acetylcholine is the primary chemical transmitter released at the neuromuscular junction. It binds to acetylcholine receptors on the muscle cell membrane, leading to muscle contraction.
Acetylcholine is the chemical that transmits signals across the neuromuscular junction. It binds to receptors on the muscle cell membrane, leading to muscle contraction.
The neurotransmitter used at the neuromuscular junction is acetylcholine. It is released from the motor neuron terminals and binds to receptors on the muscle cell membrane, leading to muscle contraction.
The transmission of the stimulus at the neuromuscular junction involves the release of acetylcholine from the motor neuron, binding to acetylcholine receptors on the muscle cell membrane, causing depolarization of the muscle cell, and ultimately leading to muscle contraction.
Acetylcholine is the primary neurotransmitter released by motor neurons at the neuromuscular junction to stimulate muscle contraction. It binds to receptors on the muscle cell membrane, initiating a series of events that ultimately lead to muscle contraction.
Β binding of acetylcholine to membrane receptors on the sarcolemma
Acetylcholine is the primary chemical transmitter released at the neuromuscular junction. It binds to acetylcholine receptors on the muscle cell membrane, leading to muscle contraction.
Acetylcholine is the chemical that transmits signals across the neuromuscular junction. It binds to receptors on the muscle cell membrane, leading to muscle contraction.
Acetylcholine
Sarcolema receptors
Acetylcholine is the primary neurotransmitter released by motor neurons at the neuromuscular junction to stimulate muscle contraction. It binds to receptors on the muscle cell membrane, initiating a series of events that ultimately lead to muscle contraction.
Blocking acetylcholine receptors can lead to a decrease in cholinergic transmission in the body. This can result in muscle weakness, altered heart rate, and impairments in cognitive function. Blocking these receptors is a mechanism of action for certain medications, such as neuromuscular blocking agents used during surgery.
Curare is a competitive inhibitor of acetylcholine. It fights with acetycholine for postsynaptic receptors.
Acetylcholine receptors are located on the motor end plate of the muscle cell membrane. This specialized region is where the nerve cell communicates with the muscle cell, allowing for the initiation of muscle contraction in response to acetylcholine binding to its receptors.
At a neuromuscular junction, the motor neuron terminal and the muscle fiber membrane (sarcolemma) approach each other. The motor neuron releases the neurotransmitter acetylcholine, which binds to receptors on the muscle fiber membrane, initiating muscle contraction.
Depolarization at the motor end plate upon arrival of action potentials triggers the release of neurotransmitter acetylcholine into the synaptic cleft. This acetylcholine then binds to receptors on the muscle cell membrane, initiating muscle contraction by depolarizing the muscle cell membrane and allowing the action potential to propagate along the muscle fiber.
The signal to excite a muscle cell involves the release of acetylcholine from the motor neuron into the synaptic cleft at the neuromuscular junction. Acetylcholine diffuses across the synaptic cleft and binds to receptors on the muscle cell membrane, leading to depolarization and muscle contraction. This process is crucial for transmitting signals from the nervous system to the muscle for movement.