A neurotransmitter that allows sodium ions to leak into a postsynaptic neuron causes excitatory postsynaptic potentials. The neurotransmitter that is not synthesized in advance and packaged into synaptic vesicles is nitric oxide.
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A synapse is a structure that allows communication between neurons. Information is transmitted across the synapse through the release of neurotransmitters from the presynaptic neuron, which then bind to receptors on the postsynaptic neuron, leading to changes in the postsynaptic neuron's electrical activity.
Neurotransmitters are chemical messengers that transmit signals and information from the presynaptic neuron to the postsynaptic neuron at the synapse. They bind to receptors on the postsynaptic neuron, leading to changes in its membrane potential and triggering a new signal to be passed along the neural pathway. Some common neurotransmitters include acetylcholine, dopamine, serotonin, and glutamate.
GABA binds to GABA receptors on the postsynaptic neuron, leading to an influx of negatively charged chloride ions into the neuron. This hyperpolarizes the neuron, making it less likely to generate an action potential and thereby inhibiting its activity.
Synaptic events refer to the processes involved in communication between neurons at synapses. These events include neurotransmitter release from the presynaptic neuron, binding of neurotransmitters to receptors on the postsynaptic neuron, and subsequent changes in the postsynaptic neuron's membrane potential. This can lead to excitation or inhibition of the postsynaptic neuron based on the type of neurotransmitter and receptors involved.
Postsynaptic potentials can be inhibitory as well. Inhibitory postsynaptic potentials (IPSPs) hyperpolarize the postsynaptic neuron, making it less likely to generate an action potential.