No, atenolol is a beta blocker that works by decreasing the heart rate and blood pressure, thereby reducing the myocardial oxygen demand.
Yes, epinephrine can decrease myocardial oxygen demand by increasing coronary perfusion, improving cardiac output, and reducing systemic vascular resistance. However, high doses of epinephrine may increase myocardial oxygen demand due to its positive inotropic and chronotropic effects.
Increased myocardial contractility increases the oxygen demand for the myocardial cells. If the demand of oxygen exceeds the supply, death of myocardial tissue can occur.
Rate-pressure product is also known as double product, which is the result of multiplying heart rate by systolic blood pressure. It is often used as an indicator of myocardial oxygen demand.
When the heart beats too rapidly, it pumps less efficiently and blood flow to the rest of the body, including the heart itself is reduced. The higher-than-normal heartbeat means there is an increase in demand for oxygen by the myocardium (heart muscle) - if this persists it can lead to myocardial infarction (heart attack), caused by the dying off of oxygen-starved myocardial cells.
increased demand for oxygen
Oxygen diffusion increases as the need increases. It is a matter of supply and demand.
There is an increase in the breathing rate, this is due to the demand for oxygen from the working muscles. •The Respiratory Volume increases, this is due to the increase of Tidal Volume and breathing rate whilst meeting the demand of oxygen for the working muscles.
The vessels that carry oxygen to the myocardial cells are called coronary arteries. These arteries supply the heart muscle with oxygen-rich blood to support its function.
Rate-pressure product is a good estimate of myocardial oxygen demand. RPP = Heart Rate X Systolic Blood Pressure. When using the RPP with patients with Coronary Artery Disease and with angina symptoms, it can indicate the oxygen demand at which Angina initially occurs (angina threshold).
The circulatory system and heart are vessels that carry oxygen to myocardial cells. They help send blood throughout the entire body each time a person's heart beats.
Increased vasoconstriction leads to an increase in afterload, which is the resistance the heart must overcome to eject blood from the left ventricle. As a result, the heart has to work harder to pump blood against the increased resistance, which can lead to increased myocardial oxygen demand and potentially contribute to the development of heart failure over time.