this process is called blood clotting...
when there is an injury , the platelets in the blood comes in contact with air they decompose to form thromboplastin, thromboplastin combine with inactive prothrombin, formed by the combination of Vitamin K and Calcium ions,to form active thrombin,thrombin combines with the fibrinogen in the blood plasma to form Fibrin, this Fibrin entangle with bolood corpuscles and thus clot is formed...
Blood clots are nature's way of keeping people from bleeding to death when they are cut or have injuries. Another term for blood clotting is coagulation. When you get a cut or puncture wound, your body's blood clotting abilities spring into action even before the first drop falls. There are several main parts of the body that act in this clotting process. The major cells responsible are the cells lining the blood vessels. The inner layers of these cells are most important in blood clot formation. People also carry a protein called fibrin. The fibrin is part of a network of proteins that stick together, with the fibrin acting like the glue in the process.
Blood clotting at first may seem like a simple drying process, but is in fact much more complex than that. By the time blood dries, a wound would have lost too much blood; and internal wounds would never stop bleeding at all as air is required for drying.
Several biological systems present in the blood interact with each other to ensure that blood forms a clot when a blood vessel is damaged, in order to close the lesion and prevent excessive bleeding. At the same time, the blood must not clot excessively as this would lead to occlusion of blood vessels and therefore death of the tissues supplied by those vessels.
When a blood vessel is injured, the clotting process has two main stages: First, haemostasis and the formation of an initial plug. Second, consolidation of the plug and formation of a durable clot.
The initial plug is formed mainly by platelets (aka thrombocytes), a cell type present in the blood which is specialised to support blood clotting. Vessel injuries expose the collagen-rich layer underneath the vessel wall, which platelets recognise and adhere to. This will activate the platelets which stick down and cause them to release a range of clotting factors. Some of these will help to capture more platelets and activate them, as well as allow them to stick to each other so that the accumulated platelets form a plug. This stops the bleeding, but the platelet plug is not very stable and the rapid blood flow underneath it will eventually tear it apart.
This is where the haemostatic system comes into play. The blood contains a range of clotting factors in their inactive forms, i.e. proteins which contribute to blood clotting (so these are not cells). The vessel injury will not only expose collagen to platelets, but also a protein known as tissue factor. This is recognised by factor VII circulating in the blood, which attaches to tissue factor and becomes activated. This sets off a chain reaction where several other clotting factors are in turn activated, until finally prothrombin is activated into thrombin. All of these proteins stick to activated membranes as they are for example found on platelets - this is how platelets help to support this chain reaction. Thrombin will then convert fibrinogen into fibrin, which forms long threads. Factor XIII then crosslinks the fibrin threads and converts them into a meshwork which surrounds and covers the platelet plug, thereby stabilising it and preventing it from rupturing. The result is a stable, solid blood clot.
This entire process takes place within 2-10 minutes in healthy individuals. Of course the blood does not stop flowing in the meantime, which means that the other blood cells (red and white) also get caught up in the clot and become a part of it. Certain white blood cells even contribute themselves towards stabilising the clot by presenting activated membranes to the clotting factors.
When your heart doesn't get enough oxygen, the muscle in the heart can feel pain. This pain is known as angina. Angina pain can be a warning that you are having a heart attack or are about to have a heart attack. However, some people have symptoms other than pain which tells them they are having low blood flow to the heart. Other symptoms can include shortness of breath, feeling tired, difficulty walking, and decreased exercise capacity.
Narrowing of the blood vessels can lead to angina or permanent heart damage which is called a heart attack.
its blood that's got alot of clot
it is a dinosaur thatoughs up blood and forms a blood clot
Quitting smoking will not cause you to suffer a blood clot. You can, of course, still have a blood clot, but it will not happen as a result of your failure to smoke.
platelets!!
A blood clot can be a circulatory disorder, if it forms in an inappropriate place. For example, a blood clot in the large veins of the legs is a circulatory disorder. In the case of a blood clot that functions to stop a broken blood vessel, the clot is part of the normal human physiology.
A thrombus is a clot that forms inside a vessel or the heart.
During clot formation, the insoluble blood protein that forms a fibrous mesh network is fibrin. This network traps blood cells and platelets to form a stable blood clot at the site of injury.
its blood that's got alot of clot
Fibrinogen is a plasma protein that is converted into fibrin, which forms the threads of a blood clot.
A thrombus is a blood clot that forms in the circulatory system and remains where it forms rather than circulating in the blood.
Bleeding occurs at the fracture site and a blood clot forms.
Allow the blood clot to resolve itself in a few weeks. If this does not happen, and the old blood clot has resulted in blurry or double vision, it is imperative to see a doctor immediately.