If 2.2 liters of gas is inhaled at 18 degrees Celsius and is heated to 38 degrees Celsius in the lungs, what is the new volume of the gas
80-100mmHg
80-100
PO2 in blood is the amount of gases in your blood. In medical terms, this is commonly called the Alveolar-arterial.
Because they are less sensitive to small decreases in arterial Po2 level.
When the aterial PO2 drops and becomes around 60 to 70 mm Hg, an increase in ventilation occurs. This is caused by a low PO2 in the blood and is detected by the carotid bodies (chemoreceptor), because the aortic and central chemoreceptors do not detect a change in arterial PO2
because it wants toExplain the way anatomical shunt through the bronchial circulation causes an PO2 difference between alveolar gas and arterial blood.? In: Circulatory System [Edit categories]
Is a mixtures of oxygenated blood with some deoxygenated blood. It results in reduction of arterial PO2 by 2mmHg and reduction of arterial oxygen saturation by 0.5% compared to oxygenated blood coming from alveolar capillaries
The PO2 does decrease in metabolic acidosis, Similarly, there is a decrease in the pH and HCO3 levels. Metabolic acidosis is a condition where the body is producing too much acid.
Although venous levels change, arterial Pco2 and Po2 levels remain surprisingly constant during exercise. In fact, Pco2 may even decline to below normal and Po2 may rise slightly because of the efficiency of the respiratory adjustments. Increased blood flow does not cause a change in gas pressures. the only way to change gas pressures is by altering atmospheric pressure, ie. scuba diving, or changing elevation. Partial pressure of any blood born gas is always directly proportional to atmospheric pressure as evidenced by Dalton's law of partial pressures. However in exercising muscle metabolic processes temporarily increase Pco2 and decrease Po2 until equalized by sufficiently oxygenated arterial blood. So the short answer to your question is that intramuscular Pco2 pressures would increase and Po2 would decrease, but the partial pressures in the blood would remain constant.
Assuming the Ka= [H+][PO2-]/[PO3-] and that PO3=PO2- then we can safely assume Ka= [H+][PO2-]/[PO2-] and so Ka= [H+][PO2-]/[PO2-] Ka=[H+] since the Ka of Phosphoric acid is equal to 7.5x10-3 then we can take -log(7.5x10-3) to find the pH=2.12
No, firstly pO2 is not a particularly good term for the measurement of oxygen within the blood as most of it is tied up in the heamoglobin molecules and as such is not part od the pO2. Secondly the pulmonary artery is the artery that carried deoxygenated blood from the heart to the lungs where they gain oxygen from the alveolar cavity. Under the laws of diffusion this means the pO2 in the alveoli must be higher than the "pO2" in the blood here, but even just common sense tells you that the oxygen levels in the blood here are very low as this is the whole point in the blood going to the lung.
Arterial po2 will not change because it's almost at maximum already. Venous po2 will decrease due to increased oxygen consumption by respiring muscle. Venous and arterial pCo2 will actually either stay the same or fall due to the increased ventilation stimulated by the increased Co2 production by respiring muscles. The increased pCO2 is detected by central and peripheral chemoreceptors and leads to increased ventilation, resulting in increased ventilation - causing pCo2 to remain normal or decrease. This mechanism cannot be used to explain the ventilation increase in light exercise because pCo2 hardly rises at all during light exercise, therefore the chemoreceptors may not be responsible for the mechanism resulting in increased ventilation,
Rather than a blood vessel with a value of 104mm Hg for Po2, it is alveolar gas thatt has a Po2 of 104 mm Hg