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because the superficial reflex fibres also come through the umn fibres along with it

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Q: Why superficial reflexes are lost in upper motor neuron lesion?
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What is the difference between an upper motor neuron lesion and a lower motor neuron lesion?

LMNLIt is due to lesion of lower motor neurons i.e. the spinal and cranial motor neurons that directly innervate the muscles.Usually single or individual muscle is affected.Disuse atrophy of muscles.All reflexes are absent.flaccid paralysis occurs.Babinski planter response not elicited.2. UMNLIt is due to upper motor neuron lesion.It involves group of muscles.spastic paralysis occurs.muscle atrophy is not severe.reflexesdeep reflexes are hyperactive.superficial refle;xes;only abdominal,cremastric and anal reflexes are lost.Babinski sign:positive.AI'IGHT,UUUMMM HOPE THIS HELPS :) LOWER MOTOR NEURON LESION: vs UPPER MOTOR NEURON LESION:1)FLACCID MUSCLE SPASTICITY OF MUSCLE2)HYPO-TONIA HYPER-TONIA3)HYPO-REFLEXIA HYPER-REFLEXIA4)PROFOUND MUSCLE ATROPHY MINIMAL MUSCLE ATROPHY5)FASCICULATIONS("TWITCHING") PRESENT FASCICULATIONS ABSNTSO JUST REMEMBER: LOWER = HYPO- EVERYTHING ; UPPER = HYPER- EVERYTHING,WELL, EXCEPT OFCOURSE - THE TWITCHING AND MUSCLE ATROPHY (ITS REVERSED).


Why hypertonicity occurs in upper motor lesion?

Hypertonicity in upper motor neuron lesions occurs due to a loss of inhibitory signals from the brain to the spinal cord, leading to increased excitatory signals and muscle tone. This results in overactivity of muscle reflexes and stiffness in the affected muscles.


What spinal tract damage tremors jerky movements?

Damage to the cerebellar pathways can result in tremors and jerky movements. Specifically, damage to the spinocerebellar pathway or cerebellar cortex can lead to uncoordinated and irregular movements. These movements are often seen in conditions such as cerebellar ataxia.


Do reflexes always involve the spinal cord?

Yes, the spinal cord controls ALL of your reflexes!


Overactive tendon reflexes with leg and arm stiffness what causes this?

This has known to be caused by Amyotrophic lateral sclerosis/ Motor Neuron disease


The spinal column controls involuntary movements called?

Spinal reflexes are involuntary movements produced by a single loop involving a sensory neuron, sometimes an intermediary neuron, and a motor neuron. A couple of examples include the deep tendon reflexes, such as the patellar reflex and the biceps reflex, and the reflex that causes one to recoil from painful stimuli, such as touching a hot surface.


What is titanus hyperreflexia?

titanus can symbolize great hyperreflexia means it overactive reflexes... Like spasm, twitching tendencies, it should be a motor neuron disease


The main symptom of upper motor neuron syndrome is?

The main symptom of upper motor neuron syndrome is spasticity, which is characterized by increased muscle tone and exaggerated reflexes. Other common symptoms include muscle weakness, difficulty with fine motor skills, and impaired voluntary movement.


Is motor a neuron?

motor neuron is a type of neuron that found at the muscles, tissues, and organs.


Reflexes involve at least three neurons and all arcs through the spinal cord?

Reflexes involve a sensory neuron that detects a stimulus, an interneuron in the spinal cord that processes the information, and a motor neuron that sends the response signal to the muscle or gland. These three neurons work together in a reflex arc to produce a rapid and involuntary response to a stimulus.


Upper motor neuron lesion?

Upper motor neuron lesions occur when there is damage to the motor pathways in the brain that control movement. Symptoms can include muscle weakness, spasticity, and hyperreflexia. Common causes include stroke, traumatic brain injury, and certain neurological conditions like multiple sclerosis.


Explain the significance of findings of brisk deep tendon reflexes and babinski sign on the affected side of samuels body?

Brisk deep tendon reflexes and a positive Babinski sign on one side of Samuel's body may indicate upper motor neuron lesion or damage, such as in conditions like stroke or spinal cord injury. These findings suggest dysfunction in the central nervous system, specifically affecting the descending motor pathways. Further evaluation is needed to determine the underlying cause and guide appropriate treatment.