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ACH receptors can be defined as an integral membrane protein that responds to the binding of acetylcholine, a neuoyansmitter. Two example are nicotinic acetyl line receptors and muscarinic acetylcholine receptors. .
Wiki User
∙ 10y ago
Wiki User
∙ 9y agoAn acetylcholine receptor is a feature of neurons which detects the presence of acetylcholine as part of neural activity.
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What statement describes nicotinic acetylcholine ACh receptors as opposed to muscarinic ACh receptors?
Nicotinic receptors are closed until ACh molecules bind to the receptor proteins.
What are the names of some common neurotransmitters?
Acetylcholine or aka ACH is the neurotransmitter that is released from the axon terminal to through the neuromuscular junction across the synaptic cleft which binds to the ACH receptors on the end motor plate of the Sarcolema.
What part of the muscle cell membrane contain acetylcholine receptors?
When the Ach binds to receptor sites on the muscle cell membrane it causes channels to open and allows Na+ to move into the cell which then causes an action potential.
Succinylcholine acts as a depolarizing agent that prevents repolarization of the nerve. Name a site within the neuromuscular junction that might be affected to prevent the muscle contraction?
It can act presynaptically on Nicotinic 2 receptors which increases the release of AcH potentiating the block at the motor end plate.
Can You List 3 Stimuli And The Receptors Used To Detect Them?
Touch - Mechanical receptors Light- Photo receptors Heat- Thermo receptors
What statement describes nicotinic acetylcholine ACh receptors as opposed to muscarinic ACh receptors?
Nicotinic receptors are closed until ACh molecules bind to the receptor proteins.
What does ach do at a neuromuscular junction?
ACh (acetylcholine) binds to receptors at the NMJ (neuromuscular junction) to induce contraction of muscle.
The first thing that stops a contraction after the nerve stops sending ACh?
AChE destroys the ACh and they are removed from the membrane receptors.
What enzyme removes ACh from receptors so stimulation of muscle cells cease?
ACh esterase, which breaks ACh down into an acetyl group and a choline group. This is then taken up by the neurone and some is reformed into ACh.
How is neostigmine chemically used to treat myasthenia gravis?
Neostigmine is a competitive irreversible inhibitor of Acetylcholinesterase (AChE), an enzyme responsible for breaking down acetylcholine (ACh). Myasthenia gravis is caused by the body producing too little ACh receptors. As stimulating the ACh receptors is needed for nervous transmission, the nerve signals cannot be transmitted causing muscle weakness and fatigue. Neostigmine is hence used to inhibit AChE so that less ACh gets broken down. This leads to more ACh binding to the ACh receptors causing muscular contraction.
What is m-cholinomimetics?
they stimulate the action of ACh at post ganglionic muscarinic receptors.
How does the incapacitating agent 3-quinuclidinyl benzilate affect the human body?
Competes with ACh at the nerve receptors and results in under stimulatin of the receptors.
How does the incapacitating agent 3-quinuclindinyl benzilate affect the human body?
Competes with ACh at the nerve receptors and results in under stimulatin of the receptors
Why does atropine effect histamine receptors?
It doesn't atropine only acts on muscarinic receptors (it's a competitive antagonist here for ACh), but histamine acts on different receptors (histamine receptors). There is no direct interaction between atropine and histamine receptors
A deficiency of ACh receptors in a motor end plate would cause and is called?
myasthenia gravis
Where are the receptors for ACH located?
At autonomic ganglia and after post ganlionic parasympathetic fibres. With few exceptions
How would a drug that stimulates acetylcholine receptors affect the sympathetic nervous system?
Preganglionic fibers for the SNS release ACh; so a drug that stimulates ACh receptors would stimulate the postganglionic fibers of sypathetic nerves, resulting in increased sympathetic activity
