Stroke volume is the volume of blood pumped out by a ventricle with each heartbeat. Yes.
Increasing heart rate does not increase stroke volume. At first, increasing exertion increases both heart rate and stroke volume. As the heart rate increases, the time spent in diastole decreases, so there is less time for the ventricles to fill with blood. The stroke volume therefore stops increasing, and as the heart rate approaches the maximum heart rate the stroke volume may begin to decrease.
stroke volume (SV) is the volume of blood pumped from one ventricle of the heart with each beat. It is calculated by subtracting the volume of blood in the ventricle at the end of a beat (called end-systolic volume) from the volume of blood just prior to the beat (called end-diastolic volume). The term stroke volume applies equally to both left and right ventricles of the heart. These two stroke volumes are generally equal, both approximately 70 ml in a healthy 70-kg man.Stroke volume is an important determinant of cardiac output, which is the product of stroke volume and heart rate. Because stroke volume decreases in certain conditions and disease states, stroke volume itself correlates with cardiac function.
Heart rate is how many times the heart contracts per minute while the stroke volume is the amount of blood ejected per beat.Cardiac output is measured by the product of both (Heart rate x stroke volume).
Back pressure exterted by arterial blood
Stroke volume is determined by three factors, altering any of them can change the stroke volume. These factors are preload, afterload, and contractility. The relationship is: SV = P*C/A What this means is that preload and contractility are directly proportional to the stroke volume and afterload is inversely proportional to stroke volume. If you increase preload (within certain limits), stroke volume will increase according to the Starling curve. Increasing contractility (many things can increase this), makes the heart pump harder and increases stroke volume. Increasing afterload decreases stroke volume. All of these can be reversed (decreasing preload and contractility = decreased stroke volume, etc). Get a good physiology book and it will explain all of this very well.
Preload, afterload, and cardiac contractility or inotropy.
Lowers stroke volume
it decreases stroke volume.its obvious!
A change in cardiac output without any change in the heart rate, pulmonary artery wedge pressure (PAWP = equated to preload) or systemic vascular resistance (SVR = afterload) would have to be due to a change in the contractility of the heart. Cardiac output (CO) is roughly equal to stroke volume x heart rate. Stroke volume is related to preload, contractility, and afterload. As you can see, the only variables you have not controlled for is cardiac contractility.
decreased stroke volume and no change in cardiac output
high blood pressure
it decreases blood volume and preload
End diastolic volume decreases which in turn affect stoke volume since sv=edv-esv