... Depends entirely on the post-synaptic neuron.
If it is an inhibitory synapse (often located on the actual body of the dendrite or soma), then the probability of that post-synaptic neuron firing DECREASES.
The reverse is true if it is excitatory; the synapse (more likely located on a dendritic spine) will more likely induce the post-synaptic neuron to fire an action potential.
When an action potential reaches the axon terminal of the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic neuron, leading to changes in its membrane potential. This process either excites or inhibits the postsynaptic neuron, depending on the neurotransmitter and receptor type involved.
neurotransmitters from the synaptic vesicles into the synapse. These neurotransmitters then bind to receptor proteins on the adjacent neuron, initiating a new action potential in the postsynaptic neuron.
Neurotransmitter receptors are located on the postsynaptic membrane of neurons. When a neurotransmitter binds to its specific receptor, it can either excite or inhibit the postsynaptic neuron, thereby influencing the transmission of signals in the brain.
Neurotransmitters can help bring another neuron to the point where it initiates an action potential by binding to postsynaptic receptor sites. If the receptors are the type that allow positively charged ions to flux through the cell membrane, and if this happens on a large enough scale (i.e., multiple sites are hit at once), then the probability of an action potential occurring becomes very high.
Basically, the release of neurotransmitters into a synaptic cleft, from an axon terminal, by causing the vesicles containing the neurotransmitters to fuse with the cell membrane, spilling their contents into the synapse.a previous answer:It can also play parts in several receptor mediated effects, these are too vast to mention them all.Search "G-Protein coupled receptors and IP3 signal cascades" in google. Might explain more.
Excitatory neurotransmitter
Neurotransmitters bind to specific proteins on the postsynaptic membrane called receptors. These receptors initiate a series of events that can either excite or inhibit the firing of the postsynaptic neuron.
When an action potential reaches the axon terminal of the presynaptic neuron, it triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic neuron, leading to changes in its membrane potential. This process either excites or inhibits the postsynaptic neuron, depending on the neurotransmitter and receptor type involved.
After the action potential reaches the presynaptic terminal, voltage-gated calcium channels open, leading to an influx of calcium ions. This triggers the release of neurotransmitters into the synaptic cleft. These neurotransmitters then bind to receptors on the postsynaptic membrane, leading to depolarization and the generation of a new action potential in the postsynaptic neuron.
neurotransmitters from the synaptic vesicles into the synapse. These neurotransmitters then bind to receptor proteins on the adjacent neuron, initiating a new action potential in the postsynaptic neuron.
Neurotransmitters diffuse across the synaptic cleft (a very short distance) and bind to receptor proteins on the postsynaptic membrane. Excitatory neurotransmitters cause sodium ions to move through receptor proteins depolarizing the membrane. Inhibitory neurotransmitters do not depolarize the postsynaptic membrane. Thus, the condition that would produce inhibition at synapse is called HYPERPOLARIZATION.
Neurotransmitter receptors are located on the postsynaptic membrane of neurons. When a neurotransmitter binds to its specific receptor, it can either excite or inhibit the postsynaptic neuron, thereby influencing the transmission of signals in the brain.
Neurotransmitters can help bring another neuron to the point where it initiates an action potential by binding to postsynaptic receptor sites. If the receptors are the type that allow positively charged ions to flux through the cell membrane, and if this happens on a large enough scale (i.e., multiple sites are hit at once), then the probability of an action potential occurring becomes very high.
A receptor potential and an excitatory postsynaptic potential (EPSP) are both graded potentials that result from the opening of ion channels in response to a stimulus. In receptor potentials, sensory receptors respond to external stimuli, leading to depolarization, while EPSPs occur when neurotransmitters bind to receptors on the postsynaptic membrane, allowing positively charged ions to flow in. Both processes can summate, contributing to the generation of action potentials if the depolarization reaches a threshold. Thus, they share mechanisms of synaptic transmission and signal transduction in the nervous system.
When a neuron is activated, there is a change in the voltage across the cell membrane at the receptor site. This change is known as a postsynaptic potential and can be either depolarizing (making the neuron more likely to fire an action potential) or hyperpolarizing (making the neuron less likely to fire an action potential).
An excitatory postsynaptic potential (EPSP) typically lasts for a few milliseconds, ranging from about 10 milliseconds to a maximum of around 50 milliseconds. The duration of an EPSP can vary depending on factors such as the specific neurotransmitter involved, the properties of the receptor, and the activity of ion channels in the postsynaptic neuron.
blockade of postsynaptic receptor