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Hypokalemia and hyperkalmia both can have effects on the heart function. Hypokalemia and hyperkalemia can cause cardiac arriythmias.
Hypokalemia potentiates the effects of digoxin. Hypokalemia reduces the drive of the Na-K-ATPase, resulting in increased cellular Na in cardiac muscles. Digoxin does the same thing by blocking the Na-K-ATPase.
Usually, hyperkalemia is associated with usage of digoxin. This is due to the blocking action of digoxin on the Na/K ase which results in accumulation of extracellular K+.Most of the times, patients presenting with heart problems are already on diuretics before they are prescribed with digoxin. Diuretics cause hypokalemia as they result in excessive excretion of K+ from the body. Hypokalemia in turn causes digoxin toxicity. Digoxin toxicity does not cause hypokalemia, but hypokalemia can worsen digoxin toxicity.
Corticosteroids have some agonistic effects on Mineralocorticoids (aldosterone) thus dumping potassium and absorbing sodium.
Weakness, Fatigue, Muscle cramps, Constipation, Abnormal heart rhythms (arrhythmias) are symptoms are hypokalemia.
Hypokalemia
Beta 2 adrenergic agonists cause increased potassium entry into cells, which can lead to hypokalemia
Hypokalemia or low potassium can cause lethal heart rhythms. general tiredness , muscle twitching and damage.
It could. If its a diabetic patient who has raised serum postassium due to diabetic nephropathy then ace inhibitor can improve his diabetic nephropathy leading to hypokalemia.... BUT it DOESNT cause hypokalemia directly... instead it leads to hyperkalemia...
Beta 2 adrenergic agonists cause increased potassium entry into cells, which can lead to hypokalemia
Hypokalamia.
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