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Yes. SD and SDHC cards are physically the same. The electronics in SDHC are just adapted so that it can hold more data in the same size (hence HC - high capacity). I am currently using an 8GB SDHC lexar media card and it works great. I can get 1200 images on JPEG fine and 600 on RAW :)

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Q: Will my camera Nikon D80 work with a SDhd card as well with as it is currently working with SD?
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Glomus tympanum tumor?

DefinitionA glomus tympanum tumor is a tumor of the middle ear and bone behind the ear (mastoid).Alternative NamesParaganglioma - glomus tympanumCauses, incidence, and risk factorsA glomus tympanum tumor grows in the temporal bone of the skull, behind the ear drum (tympanic membrane).This area contains nerve fibers (glomus bodies) that normally respond to changes in body temperature or blood pressure.These tumors usually occur late in life, around age 60 or 70, but they can appear at any age.The cause of a glomus tympanum tumor is unknown. Usually, there are no known risk factors. Glomus tumors have been associated with changes (mutations) in a gene responsible for the enzyme succinate dehydrogenase (SDHD).SymptomsHearing problems or lossRinging in the ear (pulsatile tinnitus)Weakness or loss of movement in the face (facial nerve palsy)Signs and testsGlomus tympanum tumors are diagnosed by a physical examination. They may be seen in the ear or behind the ear drum.Diagnosis also involves scans, including:CT scanMRI scanTreatmentGlomus tympanum tumors are rarely cancerous and do not tend to spread to other parts of the body. However, treatment may be needed to relieve symptoms.Expectations (prognosis)Patients who have surgery tend to do well. More than 90% of people with glomus tympanum tumors are cured.ComplicationsThe most common complication is hearing loss.Nerve damage, which may be caused by the tumor itself or damage during surgery, rarely occurs. Nerve damage can lead to facial paralysis.Calling your health care providerCall your health care provider if you notice:Difficulty with hearing or swallowingProblems with the muscles in your facePulsing sensation in your earReferencesRucker JC. Cranial neuropathies. In: Bradley WG, Daroff RB, Fenichel GM, Jankovic J. Neurology in Clinical Practice. 5th ed. Philadelphia, Pa: Saunders Elsevier;2007:chap 74.Marsh M, Jenkins H. Temporal bone neoplasms and lateral cranial base surgery. In: Cummings CW, Flint PW, Haughey BH, et al. Otolaryngology: Head & Neck Surgery. 4th ed. St. Louis, MO: Mosby;2005;chap 162.


Glomus jugulare tumor?

DefinitionA glomus jugulare tumor is a tumor of a part of the temporal bone in the skull. This tumor can affect the ear, upper neck, base of the skull, and the surrounding blood vessels and nerves.Alternative NamesParaganglioma - glomus jugulareCauses, incidence, and risk factorsA glomus jugulare tumor grows in the temporal bone of the skull, in an area called the jugular foramen. The jugular foramen is also where the jugular vein and several important nerves exit the skull.This area contains glomus bodies, which are nerve fibers that normally respond to changes in body temperature or blood pressure.These tumors usually occur later in life, around age 60 or 70, but they can appear at any age. The cause of a glomus jugulare tumor is unknown. Usually, there are no known risk factors. Glomus tumors have been associated with changes (mutations) in a gene responsible for the enzyme succinate dehydrogenase (SDHD).SymptomsSymptoms may include:Difficulty swallowing (dysphagia)DizzinessHearing problems or lossHearing pulsations in the earHoarsenessPainWeakness or loss of movement in the face (facial nerve palsy)Signs and testsGlomus jugulare tumors are diagnosed by physical examination and various scans, including:Cerebral angiographyCT scanMRI scanTreatmentGlomus jugulare tumors are rarely cancerous and do not tend to spread to other parts of the body. However, treatment may be needed to relieve symptoms. The main treatment is surgery. Surgery is complex and is usually done by both a neurosurgeon and a head and neck surgeon (neurotologist).In some cases, a procedure called embolization is performed before surgery to prevent the tumor from bleeding too much during surgery.After surgery, radiation therapy may be used to treat any part of the tumor that could not be removed completely.Some glomus tumors can be treated with stereotactic radiosurgery.Expectations (prognosis)Patients who have surgery or radiation tend to do well. More than 90% of those with glomus jugulare tumors are cured.ComplicationsThe most common complications are due to nerve damage, which may be caused by the tumor itself or damage during surgery. Nerve damage can lead to:Change in voiceDifficulty swallowingHearing lossParalysis of the faceCalling your health care providerCall your health care provider if you:Are having difficulty with hearing or swallowingDevelop pulsations in your earNotice a lump in your neckNotice any problems with the muscles in your faceReferencesRucker JC. Cranial neuropathies. In: Bradley WG, Daroff RB, Fenichel GM, Jankovic J. Neurology in Clinical Practice. 5th ed. Philadelphia, Pa: Saunders Elsevier; 2007:chap 74.Marsh M, Jenkins H. Temporal bone neoplasms and lateral cranial base surgery. In: Cummings CW, Flint PW, Haughey BH, et al. Otolaryngology: Head & Neck Surgery. 4th ed. St Louis, Mo: Mosby; 2005:chap 162.Reviewed ByReview Date: 02/01/2010David C. Dugdale, III, MD, Professor of Medicine, Division of General Medicine, Department of Medicine, University of Washington School of Medicine; and Seth Schwartz MD, MPH, Otolaryngologist, Virginia Mason Medical Center, Seattle, Washington. Also reviewed by David Zieve, MD, MHA, Medical Director, A.D.A.M., Inc.


What are the two steps in the electron transport chain producing NAPDH.?

complex I (NADH dehydrogenase, also called NADH:ubiquinone oxidoreductase; EC 1.6.5.3) removes two electrons from NADH and transfers them to a lipid-soluble carrier, ubiquinone (Q). The reduced product, ubiquinol (QH2) is free to diffuse within the membrane. At the same time, Complex I moves four protons (H+) across the membrane, producing a proton gradient. Complex I is one of the main sites at which premature electron leakage to oxygen occurs, thus being one of main sites of production of a harmful free radical called superoxide.The pathway of electrons occurs as follows:NADH is oxidized to NAD+, reducing Flavin mononucleotide to FMNH2 in one two-electron step. The next electron carrier is a Fe-S cluster, which can only accept one electron at a time to reduce the ferric ion into a ferrous ion. In a convenient manner, FMNH2 can be oxidized in only two one-electron steps, through a semiquinone intermediate. The electron thus travels from the FMNH2 to the Fe-S cluster, then from the Fe-S cluster to the oxidized Q to give the free-radical (semiquinone) form of Q. This happens again to reduce the semiquinone form to the ubiquinol form, QH2. During this process, four protons are translocated across the inner mitochondrial membrane, from the matrix to the intermembrane space. This creates a proton gradient that will be later used to generate ATP through oxidative phosphorylation.Complex II (succinate dehydrogenase; EC 1.3.5.1) is not a proton pump. It serves to funnel additional electrons into the quinone pool (Q) by removing electrons from succinate and transferring them (via FAD) to Q. Complex II consists of four protein subunits: SDHA,SDHB,SDHC, and SDHD. Other electron donors (e.g., fatty acids and glycerol 3-phosphate) also funnel electrons into Q (via FAD), again without producing a proton gradient.Complex III (cytochrome bc1 complex; EC 1.10.2.2) removes in a stepwise fashion two electrons from QH2 at the QO site and sequentially transfers them to two molecules of cytochrome c, a water-soluble electron carrier located within the intermembrane space. The two other electrons are sequentially passed across the protein to the Qi site where quinone part of ubiquinone is reduced to quinol. A proton gradient is formed because it takes 2 quinol (4H+4e-) oxidations at the Qo site to form one quinol (2H+2e-) at the Qi site. (in total 6 protons: 2 protons reduce quinone to quinol and 4 protons are released from 2 ubiquinol). The bc1 complex does NOT 'pump' protons, it helps build the proton gradient by an asymmetric absorption/release of protons.When electron transfer is hindered (by a high membrane potential, point mutations or respiratory inhibitors such as antimycin A), Complex III may leak electrons to oxygen resulting in the formation of superoxide, a highly-toxic species, which is thought to contribute to the pathology of a number of diseases, including aging.Complex IV (cytochrome c oxidase; EC 1.9.3.1) removes four electrons from four molecules of cytochrome c and transfers them to molecular oxygen (O2), producing two molecules of water (H2O). At the same time, it moves four protons across the membrane, producing a proton gradient.