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When a nerve impulse reaches an axon ending, voltage-gated calcium channels in the axonal membrane open and calcium, which is extremely low inside the cell, enters the nerve ending. The increase in calcium-ion concentration causes hundreds of synaptic vesicles to fuse with the cell membrane and expel acetylcholine into the synaptic cleft (exocytosis). The acetylcholine released at a neuromuscular junction binds reversibly to acetylcholine receptors in the muscle end-plate membrane, a postsynaptic membrane that is separated from the nerve ending by a very short distance. The receptor is a cation channel which opens when two acetylcholine molecules are bound, allowing a sodium current to enter the muscle cell and depolarize the membrane. The resulting impulse indirectly causes the muscle to contract.
Acetylcholine
acetylcholine
Cholineesterase is enzyme that hydrolyses acetylcholine .
See Acetylcholine http://en.wikipedia.org/wiki/Acetylcholine
Alpha-Motor neurons release the neurotransmitter acetylcholine at a synapse called the neuromuscular junction. When the acetylcholine binds to acetylcholine receptors on the muscle fiber, an action potential is propagated along the muscle fiber in both directions.
There are several compounds and drugs that may block acetylcholine receptors. They are collectively known as cholinergic antagonists.
acetylcholine
acetylcholine
Parasympathetic system has acetylcholine as its chemical mediator.
Acetylcholine
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Acetylcholine is the neurotransmitter at the neuromuscular junction.
acetylcholine
Cholineesterase is enzyme that hydrolyses acetylcholine .
acetylcholine (ACh)
See Acetylcholine http://en.wikipedia.org/wiki/Acetylcholine
Acetylcholine is the excitatory neurotransmitter released by neurons innervating skeletal muscles. Acetylcholine release stimulates muscle contraction by acting at the nicotinic-acetylcholine receptor on the surface of the muscle cell.